Cardiovascular disease often culminates in heart failure, a hallmark of which is fibrosis, a form of tissue scarring. Cardiac fibrosis initially repairs damaged heart tissue, but it can quickly become excessive and pathological. Identifying the mechanisms behind fibrosis is a focal point in cardiovascular research, and now, in a new study led by John W. Elrod, PhD, Director of the Aging + Cardiovascular Discovery Center and Professor of Cardiovascular Sciences at the Lewis Katz School of Medicine at Temple University, scientists at the Katz School of Medicine have discovered a critical genetic mechanism driving the process – and they have identified a novel target for reversing it. The team described their findings in a paper published online in the journal Nature Cardiovascular Research. Medical Xpress and ScienceDaily highlighted the research.
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